Dyslexia is the most common neurodevelopmental disorder, with prevalence rate ranging from 5% to 17%. What causes this disability is still somewhat uncertain, but a new theory suggests that a malfunction of the magnocellular system is responsible for behavioral deficits in dyslexia. One of the key results of the study is that the hemispheric differences in subjects with dyslexia may reflect an abnormal magnocellular development.

The prime evidence is that the number of ipsilateral connections of the lateral geniculate nucleus is less in subjects with dyslexia when compared to the control group. Also, the number of contralateral connections is greater in the test group vs the control. These studies propose a physical cause demonstrated in vivo for the development of dyslexia.

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In order to read, the cooperation between these two pathways is needed. The Parvocellular system can recognize the individual letters but it cannot quote their precise location so the magnocellular system is in charge of directing the Parvocellular system to the location of each letter so it can be identified.

Key Takeaways:

1
Dyslexia is the most common neurodevelopmental disorder with prevalence rate ranging from 5% to 17%.
2
Theory suggests that malfunction of the magnocellular system is responsible for behavioral deficits in dyslexia.
3
One of the key results of the study is that the hemispheric differences in subjects with dyslexia may reflect an abnormal magnocellular development.

The magnocellular theory of dyslexia: Monica Giraldo-Chica

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@giraldo_chica

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